The flaky and yellow skin that some people have on their faces is a sign that they may have more serious underlying conditions, according to a new study.
The researchers from the University of Calgary and the University in Beijing looked at the genetic makeup of the skin on people who had facial flaky or yellow skin, and found that some of the people with flaky facial skin had more than three times the number of mutations than people with normal skin.
The genetic changes that cause skin to appear flaky were found in people who have more mutations in the genes for collagen and epidermal growth factor, or EPIGF, than people without those genes, said Dr. Jie Yan, the lead author of the study.
Epidermal cells in the skin that make up our skin’s layer of fat and skin cells that make our faces look healthy or soft.
These cells have the ability to produce epidermis, the protective layer of skin that covers the inside of the body.
“When there is more than one mutation, it could be a sign of more serious conditions like cancer,” said Yan.
“People who have these conditions are at higher risk of developing skin cancer.”
The research team studied more than 500 people and found more than two-thirds of them had at least one of the genes that were linked to skin flakyness.
Epigenetics, the study authors explained, refers to the way that genes change over time in a gene.
A gene is an ancient, chemical blueprint that tells the cell exactly how to make proteins.
When people have a mutation in a particular gene, the cell changes in ways that increase the number or strength of the protein in the cell.
The resulting protein may have some specific function.
But mutations in other genes also affect how proteins are made and how the body works.
So, for example, if one gene is making protein that helps cells grow, the other gene may be making protein for a different purpose.
These mutations, in turn, have an effect on the way proteins are folded into a cell.
This means that the cell may look different or appear more healthy or shiny.
“It’s a genetic fingerprint that tells you about a person’s risk for developing skin diseases,” said Dr .
Jianhong Zhou, one of Yan’s co-authors and an associate professor of dermatology at the University at Buffalo.
The more mutations, the more likely the person was to develop skin flaking or yellowing.
And this pattern is repeated across the genome, which means that even if a person has no other skin conditions, the risk of skin flakiness is increased.
“We know that skin flaked skin is a real problem, but we don’t really know why, or how to fix it,” said Zhou.
He said it is possible that the skin’s natural repair mechanisms may not work, or that skin that has more mutations may be more sensitive to environmental factors like UV rays or sunburn.
“In general, people with more mutations have more skin damage.
That’s a normal process,” Zhou said.”
However, we have to be careful to take into account the effects of these mutations on the overall health of the individual.”
The researchers hope to find out whether there is a cure for skin flakeiness by studying more people, and they also want to see how people with these genetic conditions react to treatment.
The study was published in the journal Science Translational Medicine.